NURS 6501 Knowledge Check Gastrointestinal and Hepatobiliary Disorders

NURS 6501 Knowledge Check Gastrointestinal and Hepatobiliary Disorders

NURS 6501 Knowledge Check Gastrointestinal and Hepatobiliary Disorders

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In this exercise, you will complete a 10- to 20-essay type question Knowledge Check to gauge your understanding of this module’s content.

Possible topics covered in this Knowledge Check include:

  • Ulcers
  • Hepatitis markers
  • After HP shots
  • Gastroesophageal Reflux Disease
  • Pancreatitis
  • Liver failure—acute and chronic
  • Gall bladder disease
  • Inflammatory bowel disease
  • Diverticulitis
  • Jaundice
  • Bilirubin
  • Gastrointestinal bleed – upper and lower
  • Hepatic encephalopathy
  • Intra-abdominal infections (e.g., appendicitis)
  • Renal blood flow
  • Glomerular filtration rate
  • Kidney stones
  • Infections – urinary tract infections, pyelonephritis
  • Acute kidney injury
  • Renal failure – acute and chronic

ORDER NOW FOR AN ORIGINAL NURSING ESSAY ASSIGNMENT ON: NURS 6501 Knowledge Check Gastrointestinal and Hepatobiliary Disorders

Photo Credit: Getty Images

Complete the Knowledge Check By Day 7 of Week 5

To complete this Knowledge Check:

Module 3 Knowledge Check


What’s Coming Up in Module 4?

Photo Credit: [BrianAJackson]/[iStock / Getty Images Plus]/Getty Images

In Module 4, you will analyze processes related to endocrine disorders. To do this, you will analyze alterations in the relevant systems and the resultant disease processes. You will also consider patient characteristics, including racial and ethnic variables, that may impact physiological functioning and altered physiology. NURS 6501 Knowledge Check Gastrointestinal and Hepatobiliary Disorders

Week 5: Concepts of Gastrointestinal and Hepatobiliary Disorders

Patients of gastrointestinal and hepatobiliary disorders often face life-altering changes, including changes to diet, new treatment regimens, and more. For some disorders, treatments can include surgery.

Gastrointestinal conditions, such as ulcers, diverticulitis, and pancreatitis, often cause varying levels of pain and discomfort. Hepatobiliary conditions can also bring significant changes to patient routines and well-being.

This week, you examine fundamental concepts of gastrointestinal and hepatobiliary disorders. You explore common disorders in these categories, and you apply the key terms and concepts that help communicate the pathophysiological nature of these issues to patients.

Learning Objectives

Students will:

  • Analyze concepts and principles of pathophysiology across the life span

Learning Resources

Required Readings (click to expand/reduce)

McCance, K. L. & Huether, S. E. (2019). Pathophysiology: The biologic basis for disease in adults and children (8th ed.). St. Louis, MO: Mosby/Elsevier.

  • Chapter 38: Structure and Function of the Renal and Urological Systems including Summary Review
  • Chapter 39: Alteration of Renal and Urinary Function (stop at Fluids and electrolytes); Summary Review
  • Chapter 41: Structure and Function of the Digestive System (stop at Tests of digestive function); Summary Review
  • Chapter 42: Alterations of Digestive Function (stop at Cancer of the digestive track); Summary Review
Required Media (click to expand/reduce)

Module 3 Overview with Dr. Tara Harris 

Dr. Tara Harris reviews the structure of Module 3 as well as the expectations for the module. Consider how you will manage your time as you review your media and Learning Resources throughout the module to prepare for your Knowledge Check. (2m)

Liver Function Tests
Liver Diseases
Liver Pathophysiology

Online Media from Pathophysiology: The Biologic Basis for Disease in Adults and Children

In addition to this week’s media, it is highly recommended that you access and view the resources included with the course text, Pathophysiology: The Biologic Basis for Disease in Adults and Children. Focus on the videos and animations in Chapters 41 and 42 that relate to the hepatobiliary system. Refer to the Learning Resources in Week 1 for registration instructions. If you have already registered, you may access the resources at https://evolve.elsevier.com/ NURS 6501 Knowledge Check Gastrointestinal and Hepatobiliary Disorders

Week 6 Knowledge Check: Endocrine Disorders

In the Week 6 Knowledge Check, you will demonstrate your understanding of the topics covered during Module 4. This Knowledge Check will be composed of a series of questions related to specific scenarios provided. It is highly recommended that you review the Learning Resources in their entirety prior to taking the Knowledge Check, since the resources cover the topics addressed. Plan your time accordingly.

Also, during this week you will take your Midterm Exam. Please make sure to finalize and complete your Knowledge Check prior to completing your exam.

Next Module

 

  • Question 9

    1 out of 1 points

    A 65-year-old man with a history of atrial fibrillation presents to his PCP’s office 2 months after suffering from a myocardial infarction.  He declined anticoagulation due to fear he would bleed to death. He has had sudden-onset, moderately severe diffuse abdominal pain that began 18 hours ago. He has been vomiting, and he has had several episodes of diarrhea, the last of which was bloody. He has a fever of 100.9 ˚ F. CBC reveals WBC of 15,000/mm3.

    Question:

    What is the most likely mechanism behind his current symptoms?  

    Selected Answer:

    Mesenteric insufficiency particularly due to a thrombus in the superior mesenteric artery interferes with the flow of blood to the ascending colon and the small bowel. Obstructed blood flow causes mal perfusion with subsequent ischemia and infarction. In elderly patents, atherosclerosis is the primary risk factor that can cause the formation of a thrombus.

    Correct Answer:

    Thrombosis of the superior mesenteric artery, whether due to thrombotic or embolic origin, causes a sudden decrease or interruption in the primary blood flow to most of the small bowel as well as the ascending colon. This disruption leads © 2020 Walden University 6 to malperfusion of the involved end organ leading to ischemia and, ultimately, infarction. Elderly patients are at risk due to atherosclerosis which lead to thrombus formation. Embolic origins usually arise from cardiac arrythmias, usually atrial fibrillation, which leads to pooling of the blood in the atria.

    Response Feedback: [None Given]
  • Question 10

    1 out of 1 points

    A 46-year-old Caucasian female presents to the PCP’s office with a chief complaint of severe, intermittent right upper quadrant pain for the last 3 days. The pain is described as sharp and has occurred after eating french fries and cheeseburgers and radiates to her right shoulder. She has had a few episodes of vomiting “green stuff”. States had fever and chills last night which precipitated her trip to the office. She also had some dark orange urine, but she thought she was dehydrated.

    Physical exam: slightly obese female with icteric sclera as well as generalized jaundice. Temp 101˚F, pulse 108, respirations 18. Abdominal exam revealed rounded abdomen with slightly hypoactive bowel sounds. + rebound tenderness on palpation of right upper quadrant. No tenderness or rebound in epigastrium or other quadrants. Labs demonstrate elevated WBC, elevated serum alanine aminotransferase (ALT) and aspartate aminotransferase (AST) levels. Serum bilirubin (indirect) 2.5 mg/dl.  Abdominal ultrasound demonstrated enlarged gall bladder, dilated common bile duct and multiple stones in the bile duct. The APRN diagnoses the patient with acute cholecystitis and refers her to the ED for further treatment.

    Question 1 of 2:

    Describe how gallstones are formed and why they caused the symptoms that the patient presented with. 

    Selected Answer:

    The formation of gallstones occurs from impaired metabolism f bile acids, cholesterol and bilirubin. When they accumulate, they form crystals and gradually form stones. The stones can either remain dormant or begin to move in the common and cystic ducts.

    Correct Answer:

    Gallstones are formed from impaired metabolism of cholesterol, bilirubin, and bile acids. Gallstones are always composed of cholesterol, unconjugated bilirubin, bilirubin salts, fatty acids, calcium carbonates and phosphates and mucin glycoproteins. Gallstones form in bile that is supersaturated and can begin the process of cholesterol crystal formation. More crystals aggregate thus enlarging and forming stones. These stones may lie dormant or start to move down the cystic or common bile duct. These stones can cause biliary stasis, bacterial infections, biliary parasites.

    Response Feedback: [None Given]

Module 4

Results Displayed All Answers, Submitted Answers, Correct Answers, Feedback, Incorrectly Answered Questions

Question 1

1 out of 1 points

Correct

The microvilli on the intestinal brush border contain what enzymes?
Selected Answer:
Correct

oligopeptides
Answers:
Correct

oligopeptides

amino acids

gastrin

amylase
Question 2

1 out of 1 points

Correct

Cells that secrete digestive proenzymes are called?
Selected Answer:
Correct

Pancreatic acinar cells
Answers:

Kupffer cells

Correct

Pancreatic acinar cells

Hepatocytes

Parietal cells
Question 3

1 out of 1 points

Correct

The plasma BUN level is an unreliable measure of renal function because:
Selected Answer:
Correct

BUN varies because of multiple factors such as dehydration and protein intake
Answers:

BUN has no relationship to renal function

Correct

BUN varies because of multiple factors such as dehydration and protein intake

Serum sodium levels increase the BUN falsely

Serum potassium levels render the BUN inaccurate as a measure of renal function.
Question 4

1 out of 1 points

Correct

The only manifestation of cystitis in an older adult may be development of:
Selected Answer:
Correct

confusion
Answers:
Correct

confusion

headache

pelvic pain

hypertension
Question 5

1 out of 1 points

Correct

Cirrhosis and hepatitis can cause:
Selected Answer:
Correct

intrahepatic portal hypertension
Answers:
Correct

intrahepatic portal hypertension

posthepatic portal hypertension

prehepatic portal hypertension

aortic portal hypertension
Question 6

1 out of 1 points

Correct

Cells that remove bacteria and foreign particles from blood in the hepatic sinusoids are called?
Selected Answer:
Correct

Kupffer cells
Answers:
Correct

Kupffer cells

Pancreatic acinar cells

Hepatocytes

Parietal cells
Question 7

1 out of 1 points

Correct

Cells that secrete hydrochloric acid and intrinsic factor are called?
Selected Answer:
Correct

Parietal cells
Answers:

Kupffer cells

Pancreatic acinar cells

Hepatocytes

Correct

Parietal cells
Question 8

1 out of 1 points

Correct

An alkaline urinary pH significantly increases the risk for what type of urinary stone formation?
Selected Answer:
Correct

calcium phosphate
Answers:

magnesium phosphate

uric acid

Correct

calcium phosphate

potassium phosphate
Question 9

1 out of 1 points

Correct

Sympathetic stimulation of the renal arterioles causes what reaction?
Selected Answer:
Correct

vasoconstriction
Answers:

fluid excretion

diuresis

vasodilation

Correct

vasoconstriction
Question 10

1 out of 1 points

Correct

Women who have symptoms of cystitis for more than 6 weeks with negative urine cultures may have:
Selected Answer:
Correct

interstitial cystitis
Answers:

aseptic cystitis

Correct

interstitial cystitis

septic cystitis

pyelonephritis
Question 11

1 out of 1 points

Correct

A patient with sudden onset of oliguria with elevated plasma BUN and plasma creatinine levels is experiencing classic symptoms of:
Selected Answer:
Correct

Acute kidney injury
Answers:

Renal cancer

Nephrotic syndrome

Calculus lodged in ureter

Correct

Acute kidney injury
Question 12

1 out of 1 points

Correct

A gastric enzyme that digests proteins is called __________________
Selected Answer:
Correct

Pepsin
Answers:

Trypsin

Amylase

Correct

Pepsin

Ptyalin
Question 13

1 out of 1 points

Correct

The decreased renal excretion of sodium and water is caused by what substance?
Selected Answer:
Correct

Anti diuretic hormone (ADH)
Answers:
Correct

Anti diuretic hormone (ADH)

Angiotenson

Amino acids

Natriuretic peptides
Question 14

1 out of 1 points

Correct

A patient with massive proteinuria, hypoproteinemia, hyperlipidemia, and edema is experiencing classic symptoms of:
Selected Answer:
Correct

Nephrotic syndrome
Answers:

Renal cancer

Correct

Nephrotic syndrome

Calculus lodged in ureter

Acute kidney injury
Question 15

0 out of 1 points

Incorrect

Which of these structures are located in the renal cortex?
Selected Answer:
Incorrect

Glomeruli, proximal tubules, and renal corpuscles
Answers:

Glomeruli, proximal tubules, and renal corpuscles

Glomeruli, distal tubules, and pyramids

Correct

Collecting ducts, distal tubules, and pyramids

Collecting ducts, proximal tubules, and renal corpuscles
Question 16

1 out of 1 points

Correct

A pancreatic enzyme that digests proteins is called ___________________
Selected Answer:
Correct

Trypsin
Answers:
Correct

Trypsin

Amylase

Pepsin

Ptyalin
Question 17

1 out of 1 points

Correct

A patient with hematuria, dull flank pain, weight loss and anemia in the late stages of the disease are experiencing classic symptoms of:
Selected Answer:
Correct

Renal cancer
Answers:
Correct

Renal cancer

Nephrotic syndrome

Calculus lodged in ureter

Acute kidney injury
Question 18

1 out of 1 points

Correct

A patient with severe colicky flank pain radiating to the groin, nausea and vomiting, and some hematuria is experiencing classic symptoms of:
Selected Answer:
Correct

Calculus lodged in ureter
Answers:

Renal cancer

Nephrotic syndrome

Correct

Calculus lodged in ureter

Acute kidney injury
Question 19

0 out of 1 points

Incorrect

Which of these structures are located in the renal medulla?
Selected Answer:
Incorrect

Collecting ducts, proximal tubules, and renal corpuscles
Answers:

Glomeruli, proximal tubules, and renal corpuscles

Glomeruli, distal tubules, and pyramids

Correct

Collecting ducts, distal tubules, and pyramids

Collecting ducts, proximal tubules, and renal corpuscles
Question 20

1 out of 1 points

Correct

Vitamin D is activated by the kidneys and is stimulated by what hormone?
Selected Answer:
Correct

parathyroid hormone
Answers:

calcitonin

ADH

Correct

parathyroid hormone

natriuretic peptides